Normal CA1 place fields but discoordinated network discharge in a Fmr1-null mouse model of fragile X syndrome.
Circuits, Cognition & Behavior
Deregulation of synaptic plasticity in autism.
A longitudinal investigation of preferential attention to biological motion in 2- to 24-month-old infants.
Exploring rat models of 16p11.2 deletion and duplication syndromes
The rat is a more sociable species than the mouse, and a larger spectrum of behavioral assessments are available to researchers. Using outbred rat 16p11.2 deletion and duplication models recently developed in his laboratory, Yann Herault will assess whether these animal models recapitulate the mouse phenotypes. Additional communication and sociability phenotypes will also be investigated.
R-baclofen reverses cognitive deficits and improves social interactions in two lines of 16p11.2 deletion mice.
Walking, gross motor development and brain functional connectivity in infants and toddlers.
Control of recollection by slow gamma dominating mid-frequency gamma in hippocampus CA1.
Identifying specific prefrontal neurons that contribute to autism-associated abnormalities in physiology and social behavior.
Eliminating MRI motion with personalized head restraints
The short-term goal and scope of this proposal is to eliminate head motion in MRI scans at the time of acquisition by developing new, patient-specific hardware that physically prevents the head from moving in the scanner. In the longer term, Jones aims to understand the neural representation of restricted interests and how shifts in restricted interests are represented in the brain of individuals with autism.
Foxp1 in forebrain pyramidal neurons controls gene expression required for spatial learning and synaptic plasticity.
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