Molecular Mechanisms

Transcriptome analysis reveals dysregulation of innate immune response genes and neuronal activity-dependent genes in autism.

The developmental switch in GABA polarity is delayed in fragile X mice.

Snx14 regulates neuronal excitability, promotes synaptic transmission, and is imprinted in the brain of mice.

Environmental enrichment modulates 5-hydroxymethylcytosine dynamics in hippocampus.

A highly conserved program of neuronal microexons is misregulated in autistic brains.

CNTNAP4 differentially contributes to GABAergic and dopaminergic synaptic transmission.

Divergent dysregulation of gene expression in murine models of fragile x syndrome and tuberous sclerosis.

Protein interaction network of alternatively spliced isoforms from brain links genetic risk factors for autism.

Loss of Tet enzymes compromises proper differentiation of embryonic stem cells.

De novo TBR1 mutations in sporadic autism disrupt protein functions.

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