Molecular Mechanisms

TSC1 mutant neural stem/progenitor cells exhibit migration deficits and give rise to subependymal lesions in the lateral ventricle.

Centrosome motility is essential for initial axon formation in the neocortex.

Glutamate induces de novo growth of functional spines in developing cortex.

Gephyrin-mediated gamma-aminobutyric acid type A and glycine receptor clustering relies on a common binding site.

miR-132, an experience-dependent microRNA, is essential for visual cortex plasticity.

The neurexin ligands, neuroligins and leucine-rich repeat transmembrane proteins, perform convergent and divergent synaptic functions in vivo.

Neurexin-neuroligin transsynaptic interaction mediates learning-related synaptic remodeling and long-term facilitation in aplysia.

Autism-linked neuroligin-3 R451C mutation differentially alters hippocampal and cortical synaptic function.

An autism-associated point mutation in the neuroligin cytoplasmic tail selectively impairs AMPA receptor-mediated synaptic transmission in hippocampus.

Mechanisms of inhibition within the telencephalon: “where the wild things are.”

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