Molecular Mechanisms

The cell-intrinsic requirement of Sox6 for cortical interneuron development.

Mouse models of MeCP2 disorders share gene expression changes in the cerebellum and hypothalamus.

Neuroligin-2 deletion selectively decreases inhibitory synaptic transmission originating from fast-spiking but not from somatostatin-positive interneurons.

Differential gene expression in the developing lateral geniculate nucleus and medial geniculate nucleus reveals novel roles for ZIC4 and FOXP2 in visual and auditory pathway development.

LRRTM2 functions as a neurexin ligand in promoting excitatory synapse formation.

Neuroligin-1 performs neurexin-dependent and neurexin-independent functions in synapse validation.

Intrinsic patterning and experience-dependent mechanisms that generate eye-specific projections and binocular circuits in the visual pathway.

Genetic fate mapping reveals that the caudal ganglionic eminence produces a large and diverse population of superficial cortical interneurons.

Microglial activation and increased microglial density observed in the dorsolateral prefrontal cortex in autism.

Hypersensitivity to mGluR5 and ERK1/2 leads to excessive protein synthesis in the hippocampus of a mouse model of fragile X syndrome.

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