Communication impairments in mice lacking Shank1: reduced levels of ultrasonic vocalizations and scent marking behavior.
Haploinsufficiency of the autism-associated SHANK3 gene leads to deficits in synaptic function, social interaction, and social communication.
CHD8 mutation leads to autistic-like behaviors and impaired striatal circuits.
Dominant-negative SMARCA4 mutants alter the accessibility landscape of tissue-unrestricted enhancers.
R-baclofen reverses cognitive deficits and improves social interactions in two lines of 16p11.2 deletion mice.
Impaired perceptual learning in a mouse model of fragile X syndrome is mediated by parvalbumin neuron dysfunction and is reversible.
Characterization of medication use in a multicenter sample of pediatric inpatients with autism spectrum disorder.
Nucleosome turnover regulates histone methylation patterns over the genome.
Loss of the neural-specific BAF subunit ACTL6B relieves repression of early response genes and causes recessive autism.
BAF subunit switching regulates chromatin accessibility to control cell cycle exit in the developing mammalian cortex.
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