Neural coding of sex-specific social information in the mouse brain.
Quantitative gait assessment in children with 16p11.2 syndrome.
Psychotic symptoms in 16p11.2 copy-number variant carriers.
KCTD13 is a major driver of mirrored neuroanatomical phenotypes of the 16p11.2 copy number variant.
Dynamics of social representation in the mouse prefrontal cortex.
Massively parallel disruption of enhancers active during human corticogenesis.
Predictive impact of rare genomic copy number variations in siblings of individuals with autism spectrum disorders.
Abnormal auditory mismatch fields in children and adolescents with 16p11.2 deletion and 16p11.2 duplication.
Distinct pathogenic genes causing intellectual disability and autism exhibit a common neuronal network hyperactivity phenotype.
Co-localization between sequence constraint and epigenomic information improves interpretation of whole-genome sequencing data.
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