p38α MAPK signaling drives pharmacologically reversible brain and gastrointestinal phenotypes in the SERT Ala56 mouse.
Research Articles
Rbfox1 mediates cell-type-specific splicing in cortical interneurons.
A longitudinal study of parent-reported sensory responsiveness in toddlers at-risk for autism.
The severe end of the spectrum: Insights and opportunities from the Autism Inpatient Collection (AIC).
MAP1B mutations cause intellectual disability and extensive white matter deficit.
Early adolescent Rai1 reactivation reverses transcriptional and social interaction deficits in a mouse model of Smith-Magenis syndrome.
Impaired perceptual learning in a mouse model of fragile X syndrome is mediated by parvalbumin neuron dysfunction and is reversible.
Biallelic variants in VARS in a family with two siblings with intellectual disability and microcephaly: Case report and review of the literature.
CLIP and massively parallel functional analysis of CELF6 reveal a role in destabilizing synaptic gene mRNAs through interaction with 3′ UTR elements.
Altered neocortical gene expression, brain overgrowth and functional over-connectivity in Chd8 haploinsufficient mice.
- Previous Page
- Viewing
- Next Page