Genetic fate mapping reveals that the caudal ganglionic eminence produces a large and diverse population of superficial cortical interneurons.
Research Articles
A homozygous mutation in the tight-junction protein JAM3 causes hemorrhagic destruction of the brain, subependymal calcification, and congenital cataracts.
Microglial activation and increased microglial density observed in the dorsolateral prefrontal cortex in autism.
Disruption at the PTCHD1 Locus on Xp22.11 in Autism spectrum disorder and intellectual disability.
Hypersensitivity to mGluR5 and ERK1/2 leads to excessive protein synthesis in the hippocampus of a mouse model of fragile X syndrome.
Characterization of a family with rare deletions in CNTNAP5 and DOCK4 suggests novel risk loci for autism and dyslexia.
Rare familial 16q21 microdeletions under a linkage peak implicate cadherin 8 (CDH8) in susceptibility to autism and learning disability.
Early developmental regression in autism spectrum disorder: evidence from an international multiplex sample.
Does bilateral damage to the human amygdala produce autistic symptoms?
Fragile X mental retardation protein is required for synapse elimination by the activity-dependent transcription factor MEF2.
- Previous Page
- Viewing
- Next Page